Purkinje-related ventricular fibrillation associated with a homozygous H558R polymorphism in the sodium channel SCN5A gene.

نویسندگان

  • Christopher Reithmann
  • Britt-Maria Beckmann
  • Stefan Kääb
چکیده

Christopher Reithmann1*, Britt-Maria Beckmann2, and Stefan Kääb2 Medizinische Klinik I, Helios Klinikum München-West, Akademisches Lehrkrankenhaus der Universität München, Steinerweg 5, 81241 München, Germany; and Medizinische Klinik I, Klinikum Grosshadern, Ludwig-Maximilians Universität München, DZHK (German Centre for Cardiovascular Research), Partner Site Munich Heart Alliance, Munich, Germany * Corresponding author. Tel: +49 89 8892 2307; fax: +49 89 8892 2274, E-mail address: [email protected]

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SCN5A polymorphism restores trafficking of a Brugada syndrome mutation on a separate gene.

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Mutational analysis of SCN5A gene in long QT syndrome

The SCN5A gene encodes for the INa channel implicated in long QT syndrome type-3 (LQTS-type-3). Clinical symptoms of this type are lethal as most patients had a sudden death during sleep. Screening of SCN5A in South Indian cohort by PCR-SSCP analyses revealed five polymorphisms - A29A (exon-2), H558R (exon-12), E1061E and S1074R (exon-17) and IVS25 + 65G > A (exon-25) respectively. In-silico an...

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[Single nucleotide polymorphism in SCN5A and the distribution in Chinese Han ethnic group].

Mutations in voltage-gated sodium channel type (SCN5A) may evoke severe, life-threatening disturbances in cardiac rhythm, including long QT syndrome, idiopathic ventricular fibrillation (Brugada Syndrome), and isolated cardiac conduction disease. There is increasing awareness of the role of common polymorphisms in altering gene function and in susceptibility to diseases. The aim of the present ...

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Further Insights in the Most Common SCN5A Mutation Causing Overlapping Phenotype of Long QT Syndrome, Brugada Syndrome, and Conduction Defect

BACKGROUND Phenotypic overlap of type 3 long QT syndrome (LQT3), Brugada syndrome (BrS), cardiac conduction disease (CCD), and sinus node dysfunction (SND) is observed with SCN5A mutations. SCN5A-E1784K is the most common mutation associated with BrS and LQTS3. The present study examines the genotype-phenotype relationship in a large family carrying SCN5A-E1784K and SCN5A-H558R polymorphism. ...

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عنوان ژورنال:
  • Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology

دوره 18 6  شماره 

صفحات  -

تاریخ انتشار 2016